release 14.11 for windows Search Results


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Bio-Techne corporation 1411
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GL Biochem heatr1 1411–1419
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
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DeGussa Corporation damo 1411
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
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Average 90 stars, based on 1 article reviews
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Inserm Transfert inserm cic 1411
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
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BASF irganox® b 1411
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Irganox® B 1411, supplied by BASF, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
EyeTracking Inc eyetracking apparatus 1411
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Eyetracking Apparatus 1411, supplied by EyeTracking Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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DSMZ mediterranei dsm 1411
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Mediterranei Dsm 1411, supplied by DSMZ, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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93
ATCC deposit no 203021
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Deposit No 203021, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
Chemie GmbH bioproduktionssysteme
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Bioproduktionssysteme, supplied by Chemie GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/release+14%2E11+for+windows/10__1002_slash_cite__201450110-51-1-4?v=Chemie+GmbH
Average 90 stars, based on 1 article reviews
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Chemicell gmbh simag-1411 nanoparticles
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Simag 1411 Nanoparticles, supplied by Chemicell gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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NCIMB Ltd methylomicrobium alcaliphilum
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
Methylomicrobium Alcaliphilum, supplied by NCIMB Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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86
Abaqus Inc abaqus version 6 1411
<t>HEATR1</t> was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).
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Image Search Results


HEATR1 was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).

Journal: Journal of Immunology Research

Article Title: Glioma-Associated Antigen HEATR1 Induces Functional Cytotoxic T Lymphocytes in Patients with Glioma

doi: 10.1155/2014/131494

Figure Lengend Snippet: HEATR1 was overexpressed in GBM and in A2B5+GSCs. (a) qRT-PCR was performed to analyze the differential expression between GBM tissues ( n = 22) and controlled brain tissues ( n = 8). (b)-(c) IHC was performed in FFPE tissue sections of 10 primary GBM tissues (left, ×400) and 10 normal brain tissues (right, ×400). GBM tissues had higher staining score of HEATR1 protein than normal brain tissues ( P = 0.015). (d) qRT-PCR was performed to analyze the differential expression between A2B5+U87 cells and A2B5−U87 cells ( P = 0.0016).

Article Snippet: Peptides including HEATR1 2003–2011 (2003–2011, FLFDTQHFI), HEATR1 1126–1134 (1126–1134, KLLRMLFDL), HEATR1 2102–2110 (2102–2110, LLPESIPFL), HEATR1 1411–1419 (1411–1419, FLWILLILL), HEATR1 682–690 (682–670, KMVEDLISV), and HEATR1 757–765 (757–765, LMLDRGIPV) were synthesized by GL Biochem (Shanghai) Ltd. with >95% purity as indicated by analytic high-performance liquid chromatography and mass spectrometric analysis.

Techniques: Quantitative RT-PCR, Quantitative Proteomics, Staining

Binding score of  HEATR1-derived  peptides to HLA-A02 molecules.

Journal: Journal of Immunology Research

Article Title: Glioma-Associated Antigen HEATR1 Induces Functional Cytotoxic T Lymphocytes in Patients with Glioma

doi: 10.1155/2014/131494

Figure Lengend Snippet: Binding score of HEATR1-derived peptides to HLA-A02 molecules.

Article Snippet: Peptides including HEATR1 2003–2011 (2003–2011, FLFDTQHFI), HEATR1 1126–1134 (1126–1134, KLLRMLFDL), HEATR1 2102–2110 (2102–2110, LLPESIPFL), HEATR1 1411–1419 (1411–1419, FLWILLILL), HEATR1 682–690 (682–670, KMVEDLISV), and HEATR1 757–765 (757–765, LMLDRGIPV) were synthesized by GL Biochem (Shanghai) Ltd. with >95% purity as indicated by analytic high-performance liquid chromatography and mass spectrometric analysis.

Techniques: Binding Assay, Residue

HLA-A02 binding affinity of six candidate peptides. (a) Flow cytometry results of HEATR1 mix . (b) The binding activity of selected peptides to HLA-A∗02 molecule was determined semiquantitatively by measuring peptide-induced expression of HLA-A∗02 on T2 cells with flow cytometry. Data from three independent experiments were expressed as the mean ± SE. Unrelated 15-mer peptides were considered as control peptide.

Journal: Journal of Immunology Research

Article Title: Glioma-Associated Antigen HEATR1 Induces Functional Cytotoxic T Lymphocytes in Patients with Glioma

doi: 10.1155/2014/131494

Figure Lengend Snippet: HLA-A02 binding affinity of six candidate peptides. (a) Flow cytometry results of HEATR1 mix . (b) The binding activity of selected peptides to HLA-A∗02 molecule was determined semiquantitatively by measuring peptide-induced expression of HLA-A∗02 on T2 cells with flow cytometry. Data from three independent experiments were expressed as the mean ± SE. Unrelated 15-mer peptides were considered as control peptide.

Article Snippet: Peptides including HEATR1 2003–2011 (2003–2011, FLFDTQHFI), HEATR1 1126–1134 (1126–1134, KLLRMLFDL), HEATR1 2102–2110 (2102–2110, LLPESIPFL), HEATR1 1411–1419 (1411–1419, FLWILLILL), HEATR1 682–690 (682–670, KMVEDLISV), and HEATR1 757–765 (757–765, LMLDRGIPV) were synthesized by GL Biochem (Shanghai) Ltd. with >95% purity as indicated by analytic high-performance liquid chromatography and mass spectrometric analysis.

Techniques: Binding Assay, Flow Cytometry, Activity Assay, Expressing, Control

Six epitope peptides derived from the HEATR1 induce the IFN- γ response. (a) ELISpot result of 8 malignant gliomas with positive reactivity. The number of IFN- γ formingspots was calculated per 1 × 10 6 PBMCs. (b) The positive reactivity among 6 healthy donors and 38 patients only occurred in 8 malignant gliomas ( P = 0.022). GBM: glioblastoma multiforme; AA: anaplastic astrocytoma; AE: anaplastic ependymoma. This is a representative experiment from two independent experiments. No peptide stimulation was negative control. Correlation between ELISpot response and glioma grades was evaluated using a χ 2 test.

Journal: Journal of Immunology Research

Article Title: Glioma-Associated Antigen HEATR1 Induces Functional Cytotoxic T Lymphocytes in Patients with Glioma

doi: 10.1155/2014/131494

Figure Lengend Snippet: Six epitope peptides derived from the HEATR1 induce the IFN- γ response. (a) ELISpot result of 8 malignant gliomas with positive reactivity. The number of IFN- γ formingspots was calculated per 1 × 10 6 PBMCs. (b) The positive reactivity among 6 healthy donors and 38 patients only occurred in 8 malignant gliomas ( P = 0.022). GBM: glioblastoma multiforme; AA: anaplastic astrocytoma; AE: anaplastic ependymoma. This is a representative experiment from two independent experiments. No peptide stimulation was negative control. Correlation between ELISpot response and glioma grades was evaluated using a χ 2 test.

Article Snippet: Peptides including HEATR1 2003–2011 (2003–2011, FLFDTQHFI), HEATR1 1126–1134 (1126–1134, KLLRMLFDL), HEATR1 2102–2110 (2102–2110, LLPESIPFL), HEATR1 1411–1419 (1411–1419, FLWILLILL), HEATR1 682–690 (682–670, KMVEDLISV), and HEATR1 757–765 (757–765, LMLDRGIPV) were synthesized by GL Biochem (Shanghai) Ltd. with >95% purity as indicated by analytic high-performance liquid chromatography and mass spectrometric analysis.

Techniques: Derivative Assay, Enzyme-linked Immunospot, Negative Control

Single epitope peptide derived from the HEATR1 induces the IFN- γ response using ELISpot assay. PBMCs were extracted from 5 patients with HLA-A2+ GBM and 1 controlled patient with HLA-A∗02+ benign tumor. IFN- γ formingspots were calculated per 1 × 10 6 PBMC.

Journal: Journal of Immunology Research

Article Title: Glioma-Associated Antigen HEATR1 Induces Functional Cytotoxic T Lymphocytes in Patients with Glioma

doi: 10.1155/2014/131494

Figure Lengend Snippet: Single epitope peptide derived from the HEATR1 induces the IFN- γ response using ELISpot assay. PBMCs were extracted from 5 patients with HLA-A2+ GBM and 1 controlled patient with HLA-A∗02+ benign tumor. IFN- γ formingspots were calculated per 1 × 10 6 PBMC.

Article Snippet: Peptides including HEATR1 2003–2011 (2003–2011, FLFDTQHFI), HEATR1 1126–1134 (1126–1134, KLLRMLFDL), HEATR1 2102–2110 (2102–2110, LLPESIPFL), HEATR1 1411–1419 (1411–1419, FLWILLILL), HEATR1 682–690 (682–670, KMVEDLISV), and HEATR1 757–765 (757–765, LMLDRGIPV) were synthesized by GL Biochem (Shanghai) Ltd. with >95% purity as indicated by analytic high-performance liquid chromatography and mass spectrometric analysis.

Techniques: Derivative Assay, Enzyme-linked Immunospot

HEATR1-specific-peptide CTLs kill HLA-A∗02+ gliomas that express HEATR1. (a) RNA was isolated from three GBM cell lines and mRNA expression of HEATR1 was investigated by RT-PCR. (b) The values shown represent the mean ± SD of triplicate assays from PBMCs of patient number 323. U87, SHG66, and A172 were loaded with or without peptides and used as target cells in a LDH-release assay. The results showed that 6-peptides-stimulated PBMCs significantly lysed U87 and SHG66 target cells expressing both HEATR1 and HLA-A∗02 but not A172 cells that do not express HLA-A∗02 at an E/T ratio of 10 : 1. (c) Six-peptides-stimulated PBMCs from patient number 323 also significantly lysed the U87 and SHG66 GSCs at an E/T ratio of 10 : 1. Statistical differences between two groups were evaluated by the unpaired Student's t -test.

Journal: Journal of Immunology Research

Article Title: Glioma-Associated Antigen HEATR1 Induces Functional Cytotoxic T Lymphocytes in Patients with Glioma

doi: 10.1155/2014/131494

Figure Lengend Snippet: HEATR1-specific-peptide CTLs kill HLA-A∗02+ gliomas that express HEATR1. (a) RNA was isolated from three GBM cell lines and mRNA expression of HEATR1 was investigated by RT-PCR. (b) The values shown represent the mean ± SD of triplicate assays from PBMCs of patient number 323. U87, SHG66, and A172 were loaded with or without peptides and used as target cells in a LDH-release assay. The results showed that 6-peptides-stimulated PBMCs significantly lysed U87 and SHG66 target cells expressing both HEATR1 and HLA-A∗02 but not A172 cells that do not express HLA-A∗02 at an E/T ratio of 10 : 1. (c) Six-peptides-stimulated PBMCs from patient number 323 also significantly lysed the U87 and SHG66 GSCs at an E/T ratio of 10 : 1. Statistical differences between two groups were evaluated by the unpaired Student's t -test.

Article Snippet: Peptides including HEATR1 2003–2011 (2003–2011, FLFDTQHFI), HEATR1 1126–1134 (1126–1134, KLLRMLFDL), HEATR1 2102–2110 (2102–2110, LLPESIPFL), HEATR1 1411–1419 (1411–1419, FLWILLILL), HEATR1 682–690 (682–670, KMVEDLISV), and HEATR1 757–765 (757–765, LMLDRGIPV) were synthesized by GL Biochem (Shanghai) Ltd. with >95% purity as indicated by analytic high-performance liquid chromatography and mass spectrometric analysis.

Techniques: Isolation, Expressing, Reverse Transcription Polymerase Chain Reaction, Lactate Dehydrogenase Assay